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Generic nifedipine xl. (5) [nifedipine xl]-(5-bromopropyloxyphenyl)-N-[tetra(hydroxypropylmethyl)-2-oxopyrrolidin-1-yl]-1-(4-methylnitrosamino)-1H-indazole-3-carboxamide An analog of phencyclidine that is active in humans. (5-BIV-1) was added to the composition on June 5, 2015, for a period of 3 months to assess the potential of this compound to improve the efficacy of this drug in treating a neuropsychiatric condition. addition, this compound was added due to its potential for abuse. Example 1 – Pharmacology and Chemical Composition Example 1.1 Toxicity Summary Toxicity of nifedipine, both orally and intravenously, was determined. TABLE I. Toxicity of Nifedipine Dose and Administration Time of 1, 5, and 10 mg/kg orally in 6 mice, (n=8): Mice Weight (g) Oral (mg/kg) Intravenous (ml/kg) Toxicity 1, 2.5, 5, 7.5 and 10 mg/kg 3 hrs, 30 min, 1, 4, 10 6 hrs, 1.5 mg/kg (T), 1.7 (I), 1.8 (F), and 0.5 mg/kg (J) (5 days) 1, 10, 20, 30, 60 and 90 days (each) Toxicity was assessed using GSH-Px measurement, and NMR spectroscopy was used to evaluate the extent of nifedipine metabolism, and the extent of N-acyl derivative formation. GSH-Px, GSH, SOD, catalase and glutathione, the other critical antioxidants in our bodies, can help us fight pathogens, oxidative stress and tumor formation. Treatment of nifedipine was found to result in a 5 – 20 times inhibition of the rate GSH-Px production on human brain homogenates at concentrations that exceed of nifedipine's pharmacological profile in humans. The level of N-acyl derivatives, which are formed from N-ethyl-N'-nitro-N-(1-pyrroyl)carbonyl-4-methyl-N–methyl-7-hydroxy-2,3-dihydroflavone (KET-AM, 1-phenyl-2-(pyrrolidin-1-yl)phenylacetamide) are found in nifedipine. The levels of these metabolites are found to be elevated at higher doses of nifedipine in various brain tissues, including brain, cerebellum, and brainstem. The level of N-acyl derivative formation were evaluated by performing the chemical analysis of urine samples from control rats, nifedipine treated and rats with at 5, 7, and 10 mg/kg, in order to confirm whether or not the levels of these metabolites in urine may have been caused by nifedipine toxicity. The amount of metabolites found in these urine samples significantly surpassed the generic nifedipine xl levels of nifed.
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